Assistant Professor of Medicine, Massachusetts General Hospital
Pulmonology
Dr. Benjamin D. Medoff, MD
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Benjamin D. Medoff, MD works in the field of Pulmonology. He studied medicine at Harvard Medical School. Harvard Medical School is a top medical college. Harvard Medical School is ranked 1 in research and 12 in primary care medicine Benjamin D. Medoff, MD is also a published doctor. He has 52 publication published. The most recent publication is: Mechanical ventilation of the patient with chronic obstructive pulmonary disease. Doctor Benjamin D. Medoff, MD accepts Medicare payments and is listed with Medicare.gov.
Publications
- Lung T Cells in HIV Infection. Driven to Exhaustion?
- Dedifferentiation of committed epithelial cells into stem cells in vivo.
- Fat, fire and muscle--the role of adiponectin in Pulmonary vascular inflammation and remodeling.
- Compartmentalized chemokine-dependent regulatory T-cell inhibition of allergic Pulmonary inflammation.
- IL-36ñ Exerts Pro-Inflammatory Effects in the Lungs of Mice.
- Enhanced Tim3 Activity Improves Survival after Influenza Infection.
- HASH(0x9d3bf98)
- Volumetric optical frequency domain imaging of Pulmonary pathology with precise correlation to histopathology.
- CARMA1 Is Necessary for Optimal T Cell Responses in a Murine Model of Allergic Asthma.
- 18F-FDG uptake rate is a biomarker of eosinophilic inflammation and airway response in Asthma.
- Eosinophils are Necessary for Pulmonary Arterial Remodeling in a Mouse Model of Eosinophilic-Inflammation Induced Pulmonary Hypertension.
- Inhibiting CXCR3-Dependent CD8+ T Cell Trafficking Enhances Tolerance Induction in a Mouse Model of Lung Rejection.
- Interleukin-1 family member 9 stimulates chemokine production and neutrophil influx in mouse Lungs.
- Adiponectin Decreases Pulmonary Arterial Remodeling in Mouse Models of Pulmonary Hypertension.
- Adiponectin lowers glucose production by increasing SOGA.
- Case records of the Massachusetts General Hospital. Case 16-2010. A 48-year-old man with a cough and pain in the left shoulder.
- Imaging of molecular probe activity with Born-normalized fluorescence optical projection tomography.
- Adiponectin deficiency increases allergic airway inflammation and Pulmonary vascular remodeling.
- CARMA3 mediates lysophosphatidic acid-stimulated cytokine secretion by bronchial epithelial cells.
- Contribution of CCR4 and CCR8 to antigen-specific T(H)2 cell trafficking in allergic Pulmonary inflammation.
- CD11b+ myeloid cells are the key mediators of Th2 cell homing into the airway in allergic inflammation.
- The Nore1B/Mst1 complex restrains antigen receptor-induced proliferation of naïve T cells.
- Invasive and noninvasive ventilation in patients with Asthma.
- T cell trafficking in allergic Asthma: the ins and outs.
- Multiple chemokine receptors, including CCR6 and CXCR3, regulate antigen-induced T cell homing to the human Asthmatic airway.
- Oligomerization of CXCL10 is necessary for endothelial cell presentation and in vivo activity.
- CARMA1 is critical for the development of allergic airway inflammation in a murine model of Asthma.
- CXCR3 and its ligands in a murine model of obliterative bronchiolitis: regulation and function.
- STAT1 in peripheral tissue differentially regulates homing of antigen-specific Th1 and Th2 cells.
- The leukotriene B4 lipid chemoattractant receptor BLT1 defines antigen-primed T cells in humans.
- Antibody-antigen interaction in the airway drives early granulocyte recruitment through BLT1.
- Pathogenic T-cell recruitment into the airway in human Disease.
- Case 17-2005: acute chest syndrome and ARDS.
- BLT1-mediated T cell trafficking is critical for rejection and obliterative bronchiolitis after Lung transplantation.
- Case records of the Massachusetts General Hospital. Case 17-2005. A 22-year-old woman with back and leg pain and Respiratory failure.
- Leukotriene B4 receptor BLT1 mediates early effector T cell recruitment.
- Cutting edge: Th2 cell trafficking into the allergic Lung is dependent on chemoattractant receptor signaling.
- IFN-gamma-inducible protein 10 (CXCL10) contributes to airway hyperreactivity and airway inflammation in a mouse model of Asthma.
- ICOS-Expressing Lymphocytes Promote Resolution of CD8-Mediated Lung Injury in a Mouse Model of Lung Rejection.
- Differential requirement for CARMA1 in agonist-selected T-cell development.
- Variant alveolar lipoproteinosis: a syndrome with distinct clinical and pathological features.
- Development of a novel chemokine-mediated in vivo T cell recruitment assay.
- CXCR3-deficiency protects influenza-infected CCR5-deficient mice from mortality.
- Sildenafil can increase the response to inhaled nitric oxide.
- Use of recruitment maneuvers and high-positive end-expiratory pressure in a patient with acute Respiratory distress syndrome.
- Pulmonary mechanics and graphics during positive pressure ventilation.
- Patterns of vascular cell adhesion molecule-1 and intercellular adhesion molecule-1 expression in rabbit and mouse atherosclerotic lesions and at sites predisposed to ...
- Mechanical ventilation of the patient with Chronic obstructive pulmonary disease.
- Systemic oxygen extraction during incremental exercise in patients with severe Chronic obstructive pulmonary disease.
- Breathing reserve at the lactate threshold to differentiate a Pulmonary mechanical from cardiovascular limit to exercise.
- Allergic Asthma: a tale of many T cells.
Schools
Harvard Medical School
Massachusetts General Hospital
Procedures Preformed
- Bronchoscopy
- Thoracentesis
- Ventilation Management
Conditions Treated
- Acidosis
- Alkalosis
- Asthma
- Atelectasis
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
Education
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Massachusetts General Hospital -
Harvard Medical School
Hospital
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Massachusetts General Hospital
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1720062300 |
| Last Name Of The Provider | MEDOFF |
| First Name Of The Provider | BENJAMIN |
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