Assistant Professor of Medicine, Massachusetts General Hospital
Infectious Disease
Dr. Arthur Y. Kim, MD
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Arthur works in the field of Infectious Disease. He attended Harvard Medical School. Harvard Medical School is a one of the best medicals schools. Harvard Medical School is ranked 1 in research and 12 in primary care medicine Arthur Y. Kim, MD is also published. He has 53 publications published. The lastest publication: "Interferon Lambda 4 Genotype Is Associated With Jaundice and Elevated Aminotransferase Levels During Acute Hepatitis C Virus Infection: Findings From the InC3 Collabor...' He accepts Medicare.
Publications
- Treatment of hepatitis C virus-associated mixed cryoglobulinemia with direct-acting antiviral agents.
- Hepatitis C Virus Reinfection and Spontaneous Clearance of Reinfection-the InC3 Study.
- PHOTON-2: hope for patients with HIV and HCV co-infection?
- The influence of examiner type on dental students' OSCE scores.
- Genome-wide association study of hepatitis C virus- and cryoglobulin-related vasculitis.
- Admixture analysis of spontaneous hepatitis C virus clearance in individuals of African descent.
- The cost-effectiveness of improved hepatitis C virus therapies in HIV/hepatitis C virus coinfected patients.
- Hepatitis C virus serosorting in people who inject drugs: sorting out the details.
- Liver Environment and HCV Replication Affect Human T-Cell Phenotype and Expression of Inhibitory Receptors.
- An Epidemiologic Update on Hepatitis C Infection in Persons Living With or at Risk of HIV Infection.
- IL28B polymorphisms as a pretreatment predictor of response to HCV treatment.
- A simple strategy to identify acute HCV infection among newly incarcerated injection drug users.
- Broadly directed virus-specific CD4+ T cell responses are primed during acute hepatitis C infection, but rapidly disappear from human blood with viral persistence.
- Update on hepatitis C virus-specific immunity.
- Human leukocyte antigen B27 selects for rare escape mutations that significantly impair hepatitis C virus replication and require compensatory mutations.
- Compensatory Mutations Restore the Replication Defects Caused by CTL Escape Mutations in HCV Polymerase.
- Spontaneous control of HCV is associated with expression of HLA-B 57 and preservation of targeted epitopes.
- A "one-two punch" leading to hepatitis C seroconversion.
- Prospective follow-up of patients with acute hepatitis C virus infection in Brazil.
- Hepatitis C virus (HCV) sequence variation induces an HCV-specific T-cell phenotype analogous to spontaneous resolution.
- Protective effect of human leukocyte antigen B27 in hepatitis C virus infection requires the presence of a genotype-specific immunodominant CD8+ T-cell epitope.
- Interferon-induced psychosis as a "psychiatric contraindication" to hepatitis C treatment: a review and case-based discussion.
- Meta-analysis: increased mortality associated with hepatitis C in HIV-infected persons is unrelated to HIV disease progression.
- Improving the diagnosis of acute hepatitis C virus infection with expanded viral load criteria.
- Pilot study of postexposure prophylaxis for hepatitis C virus in healthcare workers.
- Rate of sustained virologic response in relation to baseline hepatitis C virus (HCV) RNA level and rapid virologic clearance in persons with acute HCV infection.
- Coinfection with HIV-1 and HCV--a one-two punch.
- Temporal dynamics of a predominant protease inhibitor-resistance mutation in a treatment-naive, hepatitis C virus-infected individual.
- Rare birds in North America: acute hepatitis C cohorts.
- Naturally occurring dominant resistance mutations to hepatitis C virus protease and polymerase inhibitors in treatment-naïve patients.
- High level of PD-1 expression on hepatitis C virus (HCV)-specific CD8+ and CD4+ T cells during acute HCV infection, irrespective of clinical outcome.
- Case records of the Massachusetts General Hospital. Case 34-2007. A 77-year-old man with ear pain, difficulty speaking, and altered mental status.
- Viral sequence evolution in acute hepatitis C virus infection.
- Sex, drugs, and hepatitis C virus in men who have sex with men: evidence for permucosal transmission.
- Immunologic evidence for lack of heterologous protection following resolution of HCV in patients with non-genotype 1 infection.
- Human leukocyte antigen-associated sequence polymorphisms in hepatitis C virus reveal reproducible immune responses and constraints on viral evolution.
- Pathogenesis of HIV-HCV Coinfection.
- Impaired hepatitis C virus-specific T cell responses and recurrent hepatitis C virus in HIV coinfection.
- Outcomes and treatment of acute hepatitis C virus infection in a United States population.
- Acute hepatitis C virus infection in incarcerated injection drug users.
- Spontaneous resolution of chronic hepatitis C virus infection: are we missing something?
- Full-breadth analysis of CD8+ T-cell responses in acute hepatitis C virus infection and early therapy.
- Broad repertoire of the CD4+ Th cell response in spontaneously controlled hepatitis C virus infection includes dominant and highly promiscuous epitopes.
- The magnitude and breadth of hepatitis C virus-specific CD8+ T cells depend on absolute CD4+ T-cell count in individuals coinfected with HIV-1.
- CD8 epitope escape and reversion in acute HCV infection.
- High resolution analysis of cellular immune responses in resolved and persistent hepatitis C virus infection.
- Predictive value of polymerase chain reaction of cerebrospinal fluid for detection of Epstein-Barr virus to establish the diagnosis of HIV-related primary central nerv...
- Comprehensive analysis of CD8(+)-T-cell responses against hepatitis C virus reveals multiple unpredicted specificities.
- The hepatitis C cascade of care: identifying priorities to improve clinical outcomes.
- Management algorithm for genotype 1 hepatitis C virus.
- Impact of new therapeutics for hepatitis C virus infection in incarcerated populations.
- Interferon Lambda 4 Genotype Is Associated With Jaundice and Elevated Aminotransferase Levels During Acute Hepatitis C Virus Infection: Findings From the InC3 Collabor...
Schools
Harvard Medical School
Massachusetts General Hospital
Doctors Specialties
- Infectious Disease Medicine
- Infectious Disease
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Blue Card PPO
- BlueConnect HMO
- Blue with Managed Care Behavioral Health HMO
- Preferred Blue PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Commercial Insurance Company
- Harvard Pilgrim Health Care PPO
- Choice Care PPO
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- Network Health
- Tufts PPO
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
Education
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Massachusetts General Hospital -
Harvard Medical School
Hospital
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Massachusetts General Hospital
Drug Facts
| NPI NUMBER | 1821089467 | |
| NPPES Provider LastName | KIM | |
| NPPES Provider FirstName | ARTHUR | |
| NPPES Provider ZIPCode | 021142621 | |
| NPPES Provider State | MA | |
| Specialty Description | Infectious Disease | |
| Total Claim Count | 294.0 | |
| Distinct Opioid Count | 1.0 | |
| Opioid Claim Count | 12.0 | |
| Percent Opioid Claims | 4.08 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1821089467 |
| Last Name Of The Provider | KIM |
| First Name Of The Provider | ARTHUR |
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