Physician
Pathology
Dr. Aron E. Lukacher, MD
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Aron Lukacher works in the field of Pathology. He graduated from Washington University School of Medicine in St. Louis. Aron Lukacher is a published doctor. He has 30 papers published. The most current publication "Pathogen Stimulation History Impacts Donor-Specific CD8(+) T Cell Susceptibility to Costimulation/Integrin Blockade-Based Therapy."
Publications
- Pathogen Stimulation History Impacts Donor-Specific CD8(+) T Cell Susceptibility to Costimulation/Integrin Blockade-Based Therapy.
- Peptide immunization elicits polyomavirus-specific MHC class ib-restricted CD8 T cells in MHC class ia allogeneic mice.
- The carboxypeptidase ACE shapes the MHC class I peptide repertoire.
- IFN-{gamma} Controls Mouse Polyomavirus Infection In Vivo.
- Some unmet challenges in the immunology of viral infections.
- Persistence of viral infection despite similar killing efficacy of antiviral CD8(+) T cells during acute and chronic phases of infection.
- Heterogeneity among viral antigen-specific CD4+ T cells and their de novo recruitment during persistent polyomavirus infection.
- Regulation of primary alloantibody response through antecedent exposure to a microbial T-cell epitope.
- Abl family tyrosine kinases regulate sialylated ganglioside receptors for polyomavirus.
- Cutting edge: shift in antigen dependence by an antiviral MHC class Ib-restricted CD8 T cell response during persistent viral infection.
- Murine Polyomavirus encodes a microRNA that cleaves early RNA transcripts but is not essential for experimental infection.
- An MHC class Ib-restricted CD8 T cell response confers antiviral immunity.
- Generation of antiviral major histocompatibility complex class I-restricted T cells in the absence of CD8 coreceptors.
- Expression of angiotensin-converting enzyme changes major histocompatibility complex class I peptide presentation by modifying C termini of peptide precursors.
- Allogeneic differences in the dependence on CD4+ T-cell help for virus-specific CD8+ T-cell differentiation.
- The antiviral CD8+ T cell response is differentially dependent on CD4+ T cell help over the course of persistent infection.
- Early virus-associated bystander events affect the fitness of the CD8 T cell response to persistent virus infection.
- Protection against polyoma virus-induced tumors is perforin-independent.
- Continuous recruitment of naive T cells contributes to heterogeneity of antiviral CD8 T cells during persistent infection.
- CD94/NKG2A expression is associated with proliferative potential of CD8 T cells during persistent polyoma virus infection.
- Costimulation requirements for antiviral CD8+ T cells differ for acute and persistent phases of polyoma virus infection.
- A cytokine promoter/yellow fluorescent protein reporter transgene serves as an early activation marker of lymphocyte subsets.
- Late priming and variability of epitope-specific CD8+ T cell responses during a persistent virus infection.
- Pathogen-host standoff: immunity to polyomavirus infection and neoplasia.
- Cutting edge: rapid in vivo CTL activity by polyoma virus-specific effector and memory CD8+ T cells.
- Memory CD8+ T cells provide an early source of IFN-gamma.
- IFN-gamma suspends the killing license of anti-tumor CTLs.
- Prevention of chronic rejection in murine cardiac allografts: a comparison of chimerism- and nonchimerism-inducing costimulation blockade-based tolerance induction reg...
- CD94-NKG2A receptors regulate antiviral CD8(+) T cell responses.
Schools
Washington Center School of Medicine
Brigham and Woman39s Hospital
Harvard Med School
Doctors Specialties
- Anatomic Pathology
- Pathology
Accepted Insurances
- Aetna
- Cigna
- Commercial Insurance Company
- Coventry Health Care
- Humana
- Medicaid
- Medicare MCR
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- United Healthcare
- Wells Fargo Insurance
Education
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Brigham and Women's Hospital -
Washington University School of Medicine in St. Louis
Hospital
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Emory University Hospital
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