Physician
Cardiology
Dr. Anthony J. Muslin, MD
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Doctor Anthony J. Muslin, MD is an expert in the field of Cardiology. He went to medical school at Harvard Medical School. Harvard Medical School is a top medical university. Harvard Medical School is ranked 1 in research and 12 in primary care medicine Anthony J. Muslin is a published MD. He has 41 papers published. The most current publication "RGS4 inhibits angiotensin II signaling and macrophage localization during renal reperfusion injury independent of vasospasm."
Publications
- RGS4 inhibits angiotensin II signaling and macrophage localization during renal reperfusion injury independent of vasospasm.
- RGS3 controls T lymphocyte migration in a model of Th2-mediated airway inflammation.
- ZnT-1 protects HL-1 cells from simulated ischemia-reperfusion through activation of Ras-ERK signaling.
- RGS4, a GTPase activator, improves renal function in ischemia-reperfusion injury.
- Akt2: a critical regulator of cardiomyocyte survival and metabolism.
- Modulation of doxorubicin-induced cardiac dysfunction in dominant-negative p38α mitogen-activated protein kinase mice.
- Phenotypic high-throughput screening in atherosclerosis research: focus on macrophages.
- Cardiovascular therapeutic discovery.
- TRB3 function in cardiac endoplasmic reticulum stress.
- Molecular basis for zinc transporter 1 action as an endogenous inhibitor of L-type calcium channels.
- New insights into cardiac aging.
- Dominant-negative p38alpha mitogen-activated protein kinase prevents cardiac apoptosis and remodeling after streptozotocin-induced diabetes mellitus.
- Serine 58 of 14-3-3zeta is a molecular switch regulating ASK1 and oxidant stress-induced cell death.
- The application of phenotypic high-throughput screening techniques to cardiovascular research.
- An unbiased chemical biology screen identifies agents that modulate uptake of oxidized LDL by macrophages.
- Uremic cardiac hypertrophy is reversed by rapamycin but not by lowering of blood pressure.
- The physician scientist training program in internal medicine at Washington University School of Medicine.
- Analysis of 14-3-3 family member function in Xenopus embryos by microinjection of antisense morpholino oligos.
- Requirement for p38 mitogen-activated protein kinase activity in neointima formation after vascular injury.
- Getting the iron out: preventing and treating heart failure in transfusion-dependent thalassemia.
- Grb2 is required for atherosclerotic lesion formation.
- Swimming stress in DN 14-3-3 mice triggers maladaptive cardiac remodeling: role of p38 MAPK.
- The 14-3-3tau phosphoserine-binding protein is required for cardiomyocyte survival.
- ATM-dependent suppression of stress signaling reduces vascular disease in metabolic syndrome.
- Akt2 regulates cardiac metabolism and cardiomyocyte survival.
- Differential role of 14-3-3 family members in Xenopus development.
- Akt1 is required for physiological cardiac growth.
- Septic shock induced from an implantable cardioverter-defibrillator lead-associated Candida albicans vegetation.
- Role of raf proteins in cardiac hypertrophy and cardiomyocyte survival.
- Differential functions of 14-3-3 isoforms in vertebrate development.
- G-protein signaling participates in the development of diabetic cardiomyopathy.
- Raf-1 kinase is required for cardiac hypertrophy and cardiomyocyte survival in response to pressure overload.
- Dominant negative 14-3-3 promotes cardiomyocyte apoptosis in early stage of type I diabetes mellitus through activation of JNK.
- Cation diffusion facilitator proteins modulate Raf-1 activity.
- Role of 14-3-3-mediated p38 mitogen-activated protein kinase inhibition in cardiac myocyte survival.
- Purification of recombinant human cPLA2 gamma and identification of C-terminal farnesylation, proteolytic processing, and carboxymethylation by MALDI-TOF-TOF analysis.
- Grb2 is required for the development of neointima in response to vascular injury.
- Hypertension and prolonged vasoconstrictor signaling in RGS2-deficient mice.
- The role of the Grb2-p38 MAPK signaling pathway in cardiac hypertrophy and fibrosis.
- Zinc ions and cation diffusion facilitator proteins regulate Ras-mediated signaling.
Schools
Harvard Medical School
Brigham Womens Hospital
University Calif
Doctors Specialties
- Cardiology
Accepted Insurances
Education
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UCSF -
Brigham and Women's Hospital -
Harvard Medical School
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