Professor, Human Growth and Development, University of Texas Southwestern Medical Center, Southwestern Medical School

Endocrinology

Andrew Zinn practices Endocrinology. He went to University of Texas Southwestern Medical School. Doctor Andrew R. Zinn, MD is also a published physician. He has 84 publication published. The most recent publication is: Evidence for a Turner syndrome locus or loci at Xp11.2-p22.1.

Publications

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• Sim1 inhibits bone formation by enhancing the sympathetic tone in male mice.
• Inducible neuronal inactivation of sim1 in adult mice causes hyperphagic obesity.
• Hyperphagia: Current concepts and future directions proceedings of the 2nd international conference on hyperphagia.
• Screening and familial characterization of copy-number variations in NR5A1 in 46,XY disorders of sex development and premature ovarian failure.
• Submicroscopic chromosomal copy number variations identified in children with hypoplastic left heart syndrome.
• UBE2A deficiency syndrome: Mild to severe intellectual disability accompanied by seizures, absent speech, urogenital, and skin anomalies in male patients.
• Unconventional wisdom about the obesity epidemic.
• A serotonin and melanocortin circuit mediates D-fenfluramine anorexia.
• Postnatal Sim1 deficiency causes hyperphagic obesity and reduced Mc4r and oxytocin expression.
• Effect of growth hormone therapy on severe short stature and skeletal deformities in a patient with combined Turner syndrome and Langer mesomelic dysplasia.
• MODY-like diabetes associated with an apparently balanced translocation: possible involvement of MPP7 gene and cell polarity in the pathogenesis of diabetes.
• An extra X or Y chromosome: contrasting the cognitive and motor phenotypes in childhood in boys with 47,XYY syndrome or 47,XXY Klinefelter syndrome.
• Cryptic chromosomal abnormalities identified in children with congenital heart disease.
• Oxytocin deficiency mediates hyperphagic obesity of Sim1 haploinsufficient mice.
• EFHC2 SNP rs7055196 is not associated with fear recognition in 45,X Turner syndrome.
• Effect of ascertainment and genetic features on the phenotype of Klinefelter syndrome.
• Cognitive and motor development during childhood in boys with Klinefelter syndrome.
• Phenotypic expansion of the supernumerary derivative (22) chromosome syndrome: VACTERL and Hirschsprung's disease.
• Dynamic regulation of p53 subnuclear localization and senescence by MORC3.
• Compound heterozygosity of SHOX-encompassing and downstream PAR1 deletions results in Langer mesomelic dysplasia (LMD).
• Increased prevalence of ADHD in Turner syndrome with no evidence of imprinting effects.
• SIM1 overexpression partially rescues agouti yellow and diet-induced obesity by normalizing food intake.
• Sim1 haploinsufficiency impairs melanocortin-mediated anorexia and activation of paraventricular nucleus neurons.
• DELETION MAPPING OF CRITICAL REGION FOR HYPOSPADIAS, PENOSCROTAL TRANSPOSITION AND IMPERFORATE ANUS ON HUMAN CHROMOSOME 13.
• Editorial: MC4R mutations--weight before screening!
• Maternal X chromosome, visceral adiposity, and lipid profile.
• A second recombination hotspot associated with SHOX deletions.
• The phenotype of short stature homeobox gene (SHOX) deficiency in childhood: contrasting children with Leri-Weill dyschondrosteosis and Turner syndrome.
• Androgen receptor CAGn repeat length influences phenotype of 47,XXY (Klinefelter) syndrome.
• X-linked reticulate pigmentary disorder with systemic manifestations: report of a third family and literature review.
• Sim1 gene dosage modulates the homeostatic feeding response to increased dietary fat in mice.
• Mesomelic and rhizomelic short stature: The phenotype of combined Leri-Weill dyschondrosteosis and achondroplasia or hypochondroplasia.
• Complete SHOX deficiency causes Langer mesomelic dysplasia.
• Coronary stent thrombosis in patients undergoing multidigit replantation.
• Voice handicap index change following treatment of voice disorders.
• Housing problems experienced by recipients of child welfare services.
• Optimizing antithrombotic strategies in patients with concomitant indications for warfarin undergoing coronary artery stenting.
• Gonadoblastoma in infancy: indications for early gonadectomy in 46XY gonadal dysgenesis.
• Giant right atrial wall vegetation mimicking cardiac tumor.
• Mutational analyses of UPIIIA, SHH, EFNB2 and HNF1beta in persistent cloaca and associated kidney malformations.
• Reassessing the cardiovascular risks and benefits of thiazolidinediones.
• Neurodevelopmental and psychosocial aspects of Turner syndrome.
• Persistent cognitive deficits in adult women with Turner syndrome.
• Early androgen deficiency in infants and young boys with 47,XXY Klinefelter syndrome.
• Cognition and the sex chromosomes: studies in Turner syndrome.
• The effect of genetic differences and ovarian failure: intact cognitive function in adult women with premature ovarian failure versus turner syndrome.
• Rps4 maps near the inactivation center on the mouse X chromosome.
• The physical phenotype of girls and women with Turner syndrome is not X-imprinted.
• A Turner syndrome neurocognitive phenotype maps to Xp22.3.
• Sequence variation at the human FOXO3 locus: a study of premature ovarian failure and primary amenorrhea.
• Refined mapping of X-linked reticulate pigmentary disorder and sequencing of candidate genes.
• Distribution and neurochemical characterization of protein kinase C-theta and -delta in the rodent hypothalamus.
• Computing power of quantitative trait locus association mapping for haploid loci.
• Human balanced translocation and mouse gene inactivation implicate Basonuclin 2 in distal urethral development.
• Clinical and molecular evaluation of SHOX/PAR1 duplications in Leri-Weill dyschondrosteosis (LWD) and idiopathic short stature (ISS).
• Phenotypes Associated with SHOX Deficiency.
• A man who inherited his SRY gene and Leri-Weill dyschondrosteosis from his mother and neurofibromatosis type 1 from his father.
• Molecular analysis of genes on Xp controlling Turner syndrome and premature ovarian failure (POF).
• The X chromosome and the ovary.
• A fork in the road to fertility.
• Assignment of microrchidia (Morc) to mouse chromosome 16 by interspecific backcross linkage analysis and human chromosome 3q13 using somatic cell hybrids and in situ h...
• The Turner syndrome-associated neurocognitive phenotype maps to distal Xp.
• Most X;autosome translocations associated with premature ovarian failure do not interrupt X-linked genes.
• Kinetics and intermediates of yeast mitochondrial DNA recombination.
• Transposition of an intron in yeast mitochondria requires a protein encoded by that intron.
• Nonreciprocal exchange between alleles of the yeast mitochondrial 21S rRNA gene: kinetics and the involvement of a double-strand break.
• Kinetic and segregational analysis of mitochondrial DNA recombination in yeast.
• In vivo double-strand breaks occur at recombinogenic G + C-rich sequences in the yeast mitochondrial genome.
• Mobile elements in the yeast mitochondrial genome.
• Inactivation of the Rps4 gene on the mouse X chromosome.
• Del (X)(p21.2) in a mother and two daughters with variable ovarian function.
• Discriminant analysis of the Ullrich-Turner syndrome neurocognitive profile.
• Growing interest in Turner syndrome.
• Prune-belly syndrome and other anomalies in a stillborn fetus with a ring X chromosome lacking XIST.
• Turner syndrome: the case of the missing sex chromosome.
• Functional equivalence of human X- and Y-encoded isoforms of ribosomal protein S4 consistent with a role in Turner syndrome.
• Structure and function of ribosomal protein S4 genes on the human and mouse sex chromosomes.
• Physical mapping of nine Xq translocation breakpoints and identification of XPNPEP2 as a premature ovarian failure candidate gene.
• Profound obesity associated with a balanced translocation that disrupts the SIM1 gene.
• New gene family defined by MORC, a nuclear protein required for mouse spermatogenesis.
• Evidence for a Turner syndrome locus or loci at Xp11.2-p22.1.
• Identification of morc (microrchidia), a mutation that results in arrest of spermatogenesis at an early meiotic stage in the mouse.
• Turner syndrome and haploinsufficiency.

Schools

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• University of Texas Southwestern Medical Center At Dallas

• Massachusetts General Hospital

• University of Texas Southwestern School of Biomedical Sciences Dean

Doctors Specialties

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• Internal Medicine
• Endocrinology

Accepted Insurances

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Education

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• Massachusetts General Hospital
• University of Texas Southwestern Medical School

Hospital

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• University of Texas Southwestern Medical Center

Drug Facts

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NPI NUMBER		1366632432
NPPES Provider LastName		ZINN
NPPES Provider FirstName		ANDREW
NPPES Provider ZIPCode		080351715
NPPES Provider State		NJ
Specialty Description		Cardiology
Total Claim Count		874.0
Distinct Opioid Count		0.0
Opioid Claim Count		0.0
Percent Opioid Claims		0.0

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Medicare Facts

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National Provider Identifier [NPI]	1366632432
Last Name Of The Provider	ZINN
First Name Of The Provider	ANDREW
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