Assistant Professor, Medicine, University of Alabama School of Medicine
Pulmonology
Dr. Amit Gaggar, MD
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Doctor Amit Gaggar, MD practices Pulmonology. He graduated from University of Alabama Medical Center. University of Alabama Medical Center is ranked 37 in research and 17 in primary care medicine Amit Gaggar is a published doctor and has 42 publications published. The lastest was: 'Toll-Like Receptor 4 Engagement Mediates Prolyl Endopeptidase Release from Airway Epithelia via Exosomes.' Amit Gaggar accepts Medicare payments and is listed with Medicare.gov.
Publications
- Toll-Like Receptor 4 Engagement Mediates Prolyl Endopeptidase Release from Airway Epithelia via Exosomes.
- Alterations in blood leukocytes of G551D-bearing cystic fibrosis patients undergoing treatment with ivacaftor.
- MMP generated matrikines.
- Exposure to cigarette smoke impacts myeloid-derived regulatory cell function and exacerbates airway hyper-responsiveness.
- Subsets of airway myeloid-derived regulatory cells distinguish mild asthma from chronic obstructive pulmonary disease.
- An Aberrant Leukotriene A4 Hydrolase-PGP Pathway in the Pathogenesis of Chronic Obstructive Pulmonary Disease.
- Pulmonary matrix metalloproteinase-9 activity in mechanically ventilated children with respiratory syncytial virus.
- L-4F Inhibits Lipopolysaccharide-Mediated Activation of Primary Human Neutrophils.
- MMP-9 Cleaves SP-D and Abrogates Its Innate Immune Functions In Vitro.
- Factors influencing the measurement of plasma/serum surfactant protein d levels by ELISA.
- Matrix metalloproteinase 9 activity enhances host susceptibility to pulmonary infection with type A and B strains of Francisella tularensis.
- Matrix metalloprotease-9 dysregulation in lower airway secretions of cystic fibrosis patients.
- The ups and downs of peer review.
- Interfering with extracellular matrix degradation to blunt inflammation.
- A novel proteolytic cascade generates an extracellular matrix-derived chemoattractant in chronic neutrophilic inflammation.
- Genetic and reproductive knowledge among adolescents and adults with cystic fibrosis.
- Potential role of high-mobility group box 1 in cystic fibrosis airway disease.
- TLR expression on neutrophils at the pulmonary site of infection: TLR1/TLR2-mediated up-regulation of TLR5 expression in cystic fibrosis lung disease.
- N-alpha-PGP and PGP, potential biomarkers and therapeutic targets for COPD.
- Neutrophils contain prolyl endopeptidase and generate the chemotactic peptide, PGP, from collagen.
- Human neutrophil elastase-mediated cleavage sites of MMP-9 and TIMP-1: implications to cystic fibrosis proteolytic dysfunction.
- Proline-Glycine-Proline (PGP) and High Mobility Group Box Protein-1 (HMGB1): Potential Mediators of Cystic Fibrosis Airway Inflammation.
- Incidence, relevance and response for Ralsfonia respiratory infections.
- Is there a doctor in the house? The downside of 24/7 attending coverage in academic intensive care units.
- A critical role for LTA4H in limiting chronic pulmonary neutrophilic inflammation.
- Attacking the multi-tiered proteolytic pathology of COPD: new insights from basic and translational studies.
- Matrix metalloproteinase activity in pediatric acute lung injury.
- The presence of a matrix-derived neutrophil chemoattractant in bronchiolitis obliterans syndrome after lung transplantation.
- Biologic markers of mortality in acute lung injury.
- A self-propagating matrix metalloprotease-9 (MMP-9) dependent cycle of chronic neutrophilic inflammation.
- Elevated levels of NO are localized to distal airways in asthma.
- Early elevation of matrix metalloproteinase-8 and -9 in pediatric ARDS is associated with an increased risk of prolonged mechanical ventilation.
- Bronchiolitis obliterans syndrome and survival following lung transplantation for patients with sarcoidosis.
- Free radical-producing myeloid-derived regulatory cells: potent activators and suppressors of lung inflammation and airway hyperresponsiveness.
- The role of matrix metalloproteinases in cystic fibrosis lung disease.
- Inhibition of airway proteases in cystic fibrosis lung disease.
- Evaluation of adenovirus vectors containing serotype 35 fibers for tumor targeting.
- Chemical transformations in individual ultrasmall biomimetic containers.
- Targeting Prolyl Endopeptidase with Valproic Acid as a Potential Modulator of Neutrophilic Inflammation.
- Anti-inflammatory mechanisms of apolipoprotein a-I mimetic Peptide in acute respiratory distress syndrome secondary to sepsis.
- Characterization and prevention of the adsorption of surfactant protein d to polypropylene.
Schools
University of Mi Med Sch
University Of Al Hospital
Conditions Treated
- Pneumonia
Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
Education
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University of Alabama Medical Center -
University of Michigan Medical School
Hospital
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University of Alabama Hospital -
Birmingham Veterans Affairs Medical Center
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1609826064 |
| Last Name Of The Provider | GAGGAR |
| First Name Of The Provider | AMIT |
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