Assistant Professor, Medicine, University of Chicago, Pritzker School of Medicine
Endocrinology
Dr. Alexandra M. Dumitrescu, MD
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Alexandra Dumitrescu works in the field of Endocrinology. He has 1 awards "CMS Meaningful Use Stage 1 Certification". Alexandra Dumitrescu is a published doctor and has 45 publications published. The lastest was: 'The Thyroid Hormone Analog DITPA Ameliorates Metabolic Parameters of Male Mice With Mct8 Deficiency.' Alexandra Dumitrescu accepts Medicare payments and is registered with Medicare.gov.
Publications
- The Thyroid Hormone Analog DITPA Ameliorates Metabolic Parameters of Male Mice With Mct8 Deficiency.
- A novel mechanism of inherited TBG Deficiency: mutation in a liver-specific enhancer.
- Placenta Passage of the Thyroid Hormone Analog DITPA to Male Wild-Type and Mct8-Deficient Mice.
- Classification and proposed nomenclature for inherited defects of Thyroid hormone action, cell transport, and metabolism.
- Inherited defects in Thyroid hormone cell-membrane transport and metabolism.
- A Novel Mutation in the Albumin Gene (R218S) causing Familial Dysalbuminemic Hyperthyroxinemia in a Family of Bangladeshi Extraction.
- Inherited defects of Thyroid hormone-cell-membrane transport: review of recent findings.
- Mct8 deficient mice have increased energy expenditure and reduced fat mass that is abrogated by normalization of serum T3 levels.
- The syndromes of reduced sensitivity to Thyroid hormone.
- Incidental Identification of a Thyroid Hormone Receptor beta (THRB) Gene Variant in a Family with Autoimmune Thyroid Disease.
- Changes in Thyroid Status During Perinatal Development of MCT8-Deficient Male Mice.
- Diiodothyropropionic Acid (DITPA) in the Treatment of MCT8 Deficiency.
- Consecutive Mutational Events in a Thyroid Stimulating Hormone (TSH) Receptor Allele of Arab Families with Resistance to TSH.
- The Coexistence of a Novel Inactivating Mutant Thyrotropin Receptor Allele with Two Thyroid Peroxidase Mutations: A Genotype-Phenotype Correlation.
- Distinct roles of deiodinases on the phenoType of Mct8 defect: a comparison of eight different mouse genoTypes.
- Mice deficient in MCT8 reveal a mechanism regulating Thyroid hormone secretion.
- Thyroid hormone-regulated mouse cerebral cortex genes are differentially dependent on the source of the hormone: a study in monocarboxylate transporter-8- and deiodina...
- The syndrome of inherited partial SBP2 Deficiency in humans.
- A Thyroid hormone analog with reduced dependence on the monocarboxylate transporter 8 for tissue transport.
- Importance of monocarboxylate transporter 8 for the blood-brain barrier-dependent availability of 3,5,3'-triiodo-L-thyronine.
- Selenium supplementation fails to correct the selenoprotein synthesis defect in subjects With SBP2 gene mutations.
- A novel monocarboxylate transporter 8 gene mutation as a cause of severe neonatal hypotonia and developmental delay.
- Mosaicism of a Thyroid hormone receptor-beta gene mutation in resistance to Thyroid hormone.
- Tissue-specific Thyroid hormone deprivation and excess in monocarboxylate transporter (mct) 8-deficient mice.
- Thyroid hormone mediated changes in gene expression can be initiated by cytosolic action of the Thyroid hormone receptor beta through the phosphatidylinositol 3-kinase...
- Cytosolic action of Thyroid hormone leads to induction of hypoxia-inducible factor-1alpha and glycolytic genes.
- Mutations in SECISBP2 result in Abnormal thyroid hormone metabolism.
- A de novo mutation in an already mutant nucleotide of the Thyroid hormone receptor beta gene perpetuates resistance to Thyroid hormone.
- Thyroid hormone responsive genes in cultured human fibroblasts.
- A novel syndrome combining thyroid and neurological Abnormalities is associated with mutations in a monocarboxylate transporter gene.
- Novel biological and clinical aspects of Thyroid hormone metabolism.
- Syndromes of reduced sensitivity to Thyroid hormone: genetic defects in hormone receptors, cell transporters and deiodination.
- X-linked paroxysmal dyskinesia and severe global retardation caused by defective MCT8 gene.
- Approach to the patient with resistance to Thyroid hormone and pregnancy.
- Pituitary-Thyroid setpoint and thyrotropin receptor expression in consomic rats.
- A new case of resistance to Thyroid hormone caused by a de novo P453T mutation in the Thyroid hormone receptor gene in an Israeli child.
- Inherited defects of Thyroid hormone metabolism.
- Partial deficiency of Thyroid transcription factor 1 produces predominantly neurological defects in humans and mice.
- Congenital secondary hypoThyroidism caused by exon skipping due to a homozygous donor splice site mutation in the TSHbeta-subunit gene.
- Complete thyroxine-binding globulin (TBG) deficiency in two families Without mutations in coding or promoter regions of the TBG genes: in vitro demonstration of exon s...
- A case of Resistance to Thyroid Hormone without mutation in the Thyroid hormone receptor beta.
- Stanniocalcin 1 induction by Thyroid hormone depends on Thyroid hormone receptor β and phosphatidylinositol 3-kinase activation.
- Role of Type 2 deiodinase in response to acute lung injury (ALI) in mice.
- Cerebral cortex hyperThyroidism of newborn mct8-deficient mice transiently suppressed by lat2 inactivation.
Conditions Treated
- Abnormal Thyroid
- Calcium Metabolism Disorders
- Diabetes Mellitus, Secondary
- Diabetes Type 1
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Doctors Specialties
- Endocrinology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Blue Card PPO
- Blue Cross Blue Shield of Illinois PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Cofinity PPO
- Coventry Health Care of Illinois PPO
- Health and Life PPO
- Platinum PPO
- First Health (Coventry Health Care) PPO
- HealthLink PPO
- MultiPlan PPO
- PHCS Network PPO
- Sagamore Plus
- Choice Plus POS
- Options PPO
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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University of Chicago -
Carol Davila University of Medicine and Pharmacy
Hospital
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University of Chicago Medical Center
Drug Facts
| NPI NUMBER | 1003078049 | |
| NPPES Provider LastName | DUMITRESCU | |
| NPPES Provider FirstName | ALEXANDRA | |
| NPPES Provider ZIPCode | 606371447 | |
| NPPES Provider State | IL | |
| Specialty Description | Endocrinology | |
| Total Claim Count | 714.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1003078049 |
| Last Name Of The Provider | DUMITRESCU |
| First Name Of The Provider | ALEXANDRA |
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