Assoc Prof in Microbiology-Immunology and Medicine-Infectious Diseases, Northwestern University Medical School

Infectious Disease

Alan Hauser is an expert in the field of Infectious Disease. He graduated from University of Minnesota Medical School. University of Minnesota Medical School has a rank of 40 in research and a 10 in primary care Dr. Alan R. Hauser is also a published doctor. He has 53 paper published. The most recent paper is: Another toxic shock syndrome. Streptococcal infection is even more dangerous than the staphylococcal form. He accepts all Medicare patients.

Publications

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• Utility of Whole-Genome Sequencing in Characterizing Acinetobacter Epidemiology and Analyzing Hospital Outbreaks.
• The Pseudomonas aeruginosa Type III Translocon Is Required for Biofilm Formation at the Epithelial Barrier.
• Influence of ACB complex genospecies on clinical outcomes in a U.S. hospital with high rates of multidrug resistance.
• Different paths to pathogenesis.
• Bacterial and Clinical Characteristics of Health Care- and Community-Acquired Bloodstream Infections Due to Pseudomonas aeruginosa.
• Structure of the Type III Secretion Effector Protein ExoU in Complex with Its Chaperone SpcU.
• Draft genome sequence of the Pseudomonas aeruginosa bloodstream isolate PABL056.
• An rhs gene of Pseudomonas aeruginosa encodes a virulence protein that activates the inflammasome.
• Clinical outcomes of type III Pseudomonas aeruginosa bacteremia.
• Pseudomonas aeruginosa: So many virulence factors, so little time.
• Clinical significance of microbial infection and adaptation in cystic fibrosis.
• The accessory genome of Pseudomonas aeruginosa.
• Role of the membrane localization domain of the Pseudomonas aeruginosa effector protein ExoU in cytotoxicity.
• Pseudomonas aeruginosa cytotoxin ExoU is injected into phagocytic cells during acute pneumonia.
• Morbidity associated with Pseudomonas aeruginosa bloodstream infections.
• Pseudomonas aeruginosa virulence and therapy: evolving translational strategies.
• Evolution of Pseudomonas aeruginosa type III secretion in cystic fibrosis: a paradigm of chronic infection.
• Hybrid pathogenicity island PAGI-5 contributes to the highly virulent phenotype of a Pseudomonas aeruginosa isolate in mammals.
• Pseudomonas aeruginosa induces localized immunosuppression during pneumonia.
• Pseudomonas aeruginosa: an uninvited guest refuses to leave.
• A C-terminal domain targets the Pseudomonas aeruginosa cytotoxin ExoU to the plasma membrane of host cells.
• Functional regions of the Pseudomonas aeruginosa cytotoxin ExoU.
• Severe Pseudomonas aeruginosa infections. Tackling the conundrum of drug resistance.
• Relative contributions of Pseudomonas aeruginosa ExoU, ExoS, and ExoT to virulence in the lung.
• Type III secretion phenotypes of Pseudomonas aeruginosa strains change during infection of individuals with cystic fibrosis.
• Survey of ferroxidase expression and siderophore production in clinical isolates of Pseudomonas aeruginosa.
• Secretion of the toxin ExoU is a marker for highly virulent Pseudomonas aeruginosa isolates obtained from patients with hospital-acquired pneumonia.
• Pseudomonas aeruginosa ExoU, a toxin transported by the type III secretion system, kills Saccharomyces cerevisiae.
• Subinhibitory bismuth-thiols reduce virulence of Pseudomonas aeruginosa.
• Type III protein secretion is associated with poor clinical outcomes in patients with ventilator-associated pneumonia caused by Pseudomonas aeruginosa.
• Characterization of the core and accessory genomes of Pseudomonas aeruginosa using bioinformatic tools Spine and AGEnt.
• Draft Genome Sequence of Acinetobacter baumannii Strain ABBL099, a Multidrug-Resistant Clinical Outbreak Isolate with a Novel Multilocus Sequence Type.
• Type IV collagen-mediated melanoma cell adhesion and migration: involvement of multiple, distinct domains of the collagen molecule.
• Another toxic shock syndrome. Streptococcal infection is even more dangerous than the staphylococcal form.
• Cloning of the gene, speB, for streptococcal pyrogenic exotoxin type B in Escherichia coli.
• Streptococcus pyogenes causing toxic-shock-like syndrome and other invasive diseases: clonal diversity and pyrogenic exotoxin expression.
• Molecular analysis of pyrogenic exotoxins from Streptococcus pyogenes isolates associated with toxic shock-like syndrome.
• Nucleotide sequence of the streptococcal pyrogenic exotoxin type B gene and relationship between the toxin and the streptococcal proteinase precursor.
• Identification of Pseudomonas aeruginosa genes required for epithelial cell injury.
• PepA, a secreted protein of Pseudomonas aeruginosa, is necessary for cytotoxicity and virulence.
• Defects in type III secretion correlate with internalization of Pseudomonas aeruginosa by epithelial cells.
• Pseudomonas aeruginosa induces type-III-secretion-mediated apoptosis of macrophages and epithelial cells.
• Pseudomonas aeruginosa gene products PilT and PilU are required for cytotoxicity in vitro and virulence in a mouse model of acute pneumonia.
• The arginine finger domain of ExoT contributes to actin cytoskeleton disruption and inhibition of internalization of Pseudomonas aeruginosa by epithelial cells and mac...
• The biofilm life cycle and virulence of Pseudomonas aeruginosa are dependent on a filamentous prophage.
• Prevalence of type III secretion genes in clinical and environmental isolates of Pseudomonas aeruginosa.
• Interactions between effector proteins of the Pseudomonas aeruginosa type III secretion system do not significantly affect several measures of disease severity in mamm...
• Genomic islands of Pseudomonas aeruginosa.
• Pseudomonas aeruginosa uses type III secretion system to kill biofilm-associated amoebae.
• Phosphorylation-independent interaction between 14-3-3 and exoenzyme S: from structure to pathogenesis.
• The type III secretion system of Pseudomonas aeruginosa: infection by injection.
• Electrostatic interactions play a minor role in the binding of ExoS to 14-3-3 proteins.

Schools

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• University of Mn Med Sch

• University Of Minnesota

• University Calif San Francisco

Doctors Specialties

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• Infectious Disease Medicine
• Infectious Disease

Accepted Insurances

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Education

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• UCSF
• University of Minnesota at Fairview
• University of Minnesota Medical School

Hospital

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• Northwestern Memorial Hospital
• Jesse Brown Veterans Affairs Medical Center

Helpful Reviews

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Medicare Facts

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National Provider Identifier [NPI]	1790881647
Last Name Of The Provider	HAUSER
First Name Of The Provider	ALAN
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