Assistant Professor, Dermatology, Perelman School of Medicine

Dermatology

Aimee Payne practices Dermatology. She attended Washington University School of Medicine in St. Louis. She received 1 awards: "CMS Meaningful Use Stage 1 Certification". Aimee Payne has written publications. She has 40 publications published with PubMed. The lastest publication is: 'Functional expression of the Wilson disease protein reveals mislocalization and impaired copper-dependent trafficking of the common H1069Q mutation.' She is registered with Medicare and accepts Medicare payments.

Publications

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• Reliability and Validity of Cutaneous Sarcoidosis Outcome Instruments Among Dermatologists, Pulmonologists, and Rheumatologists.
• Subcutaneous Veltuzumab, a Humanized Anti-CD20 Antibody, in the Treatment of Refractory Pemphigus Vulgaris.
• The dual nature of interleukin-10 in Pemphigus vulgaris.
• Persistence of Anti-Desmoglein 3 IgG(+) B-Cell Clones in Pemphigus Patients over Years.
• Plakophilins, desmogleins, and Pemphigus: the tail wagging the dog.
• Reliability and convergent validity of the cutaneous sarcoidosis activity and morphology instrument for assessing cutaneous sarcoidosis.
• MAPKAP kinase 2 (MK2)-dependent and independent models of blister formation in Pemphigus vulgaris.
• Pediatric Pemphigus Vulgaris: Durable Treatment Responses Achieved with Prednisone and Mycophenolate Mofetil (MMF).
• Enrichment of total serum IgG4 in Pemphigus patients.
• Rituximab for autoimmune blistering diseases: recent studies, new insights.
• Inhibitory human antichimeric antibodies to rituximab in a patient with Pemphigus.
• Definitions and outcome measures for bullous Pemphigoid: Recommendations by an international panel of experts.
• The autoimmune regulator (AIRE), which is defective in autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy patients, is expressed in human epidermal and fol...
• p38 MAPK activation is downstream of the loss of interCellular adhesion in pemphigus vulgaris.
• Autoimmunity to desmocollin 3 in Pemphigus vulgaris.
• Cleavage isn't everything: potential novel mechanisms of exfoliative toxin-mediated blistering.
• Homologous regions of autoantibody heavy chain complementarity-determining region 3 (H-CDR3) in patients with Pemphigus cause pathogenicity.
• No evidence of Skin blisters with human desmocollin-3 gene mutation.
• Antibodies to the desmoglein 1 precursor proprotein but not to the mature Cell surface protein cloned from individuals without pemphigus.
• Reliability and convergent validity of two outcome instruments for Pemphigus.
• Pathogenic anti-desmoglein MAbs show variable ELISA activity because of preferential binding of mature versus proprotein isoforms of desmoglein 3.
• DeltaNp63 knockdown mice: A mouse model for AEC syndrome.
• Disruption of desmosome assembly by monovalent human Pemphigus vulgaris monoclonal antibodies.
• Consensus statement on definitions of disease, end points, and therapeutic response for Pemphigus.
• Targeting Pemphigus autoantibodies through their heavy-chain variable region genes.
• Cloning and genetic characterization of human Pemphigus autoantibodies.
• Dermatologic toxicity of chemotherapeutic agents.
• Two novel TP63 mutations associated with the ankyloblepharon, ectodermal defects, and cleft lip and palate syndrome: a Skin fragility phenotype.
• Genetic and functional characterization of human Pemphigus vulgaris monoclonal autoantibodies isolated by phage display.
• Desmosomes and disease: pemphigus and bullous Impetigo.
• The role of chemokines in melanoma tumor growth and metastasis.
• Shared VH1-46 gene usage by Pemphigus vulgaris autoantibodies indicates common humoral immune responses among patients.
• Update on the cloning of monoclonal anti-desmoglein antibodies from human Pemphigus patients: implications for targeted therapy.
• Seeking approval: present and future therapies for Pemphigus vulgaris.
• Beyond steric hindrance: the role of adhesion signaling pathways in the pathogenesis of Pemphigus.
• Efficacy of intravenous immunoglobulin (IVIG) affinity-purified anti-desmoglein anti-idiotypic antibodies in the treatment of an experimental model of Pemphigus vulgaris.
• Ajuba, a novel LIM protein, interacts with Grb2, augments mitogen-activated protein kinase activity in fibroblasts, and promotes meiotic maturation of Xenopus oocytes ...
• Functional expression of the menkes disease protein reveals common biochemical mechanisms among the copper-transporting P-type ATPases.
• Functional expression of the Wilson disease protein reveals mislocalization and impaired copper-dependent trafficking of the common H1069Q mutation.

Schools

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• Washington University In St Louis School Of Medicine

Procedures Preformed

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• Destruction of Benign Skin Lesion
• Destruction of Malignant Skin Lesion
• Excision of Benign Skin Lesion

Conditions Treated

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• Acne
• Actinic Keratosis
• Athlete's Foot
• Basal Cell Carcinoma
• View All

Doctors Specialties

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• Dermatology

Accepted Insurances

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Awards

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• CMS Meaningful Use Stage 1 Certification

Education

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• University of Pennsylvania Health System
• Pennsylvania Hospital - University of Pennsylvania Health System
• Washington University School of Medicine in St. Louis

Hospital

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• Penn Presbyterian Medical Center
• Pennsylvania Hospital
• Hospital of the University of Pennsylvania

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