Physician
Neurology
Dr. Abdolmohamad Rostami, MD
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Doctor Abdolmohamad Rostami, MD is an expert in the field of Neurology. He received awards:"CMS Meaningful Use Stage 1 Certification". Abdolmohamad Rostami is a published doctor and has 98 publications published. The lastest was: 'Free light chains in multiple sclerosis urine.'
Publications
- Apoptotic cell-treated dendritic cells induce immune tolerance by specifically inhibiting development of CD4(+) effector memory T cells.
- IL-12Rβ2 has a protective role in relapsing-remitting experimental autoimmune encephalomyelitis.
- Emerging immunopharmacological targets in Multiple sclerosis.
- c-kit plays a critical role in induction of intravenous tolerance in experimental autoimmune encephalomyelitis.
- Analysis of 13 cell types reveals evidence for the expression of numerous novel primate- and tissue-specific microRNAs.
- Silencing IFN-γ Binding/Signaling in Astrocytes versus Microglia Leads to Opposite Effects on Central Nervous System Autoimmunity.
- 1,25-Dihydroxyvitamin D3 enhances neural stem cell proliferation and oligodendrocyte differentiation.
- Immunotherapy using lipopolysaccharide-stimulated bone marrow-derived dendritic cells to treat experimental autoimmune encephalomyelitis.
- Committed Tc17 cells are phenotypically and functionally resistant to the effects of IL-27.
- Astrocyte-derived lactosylceramide implicated in Multiple sclerosis.
- Tolerogenic dendritic cells produced by Lentiviral-mediated CD40- and IL-23p19-specific shRNA can ameliorate experimental autoimmune encephalomyelitis by suppressing T...
- Neurotrophin 3 Transduction Augments Remyelinating and Immunomodulatory Capacity of Neural Stem Cells.
- Anti-MS4a4B treatment abrogates MS4a4B-mediated protection in T cells and ameliorates experimental autoimmune encephalomyelitis.
- DAB389IL-2 suppresses autoimmune inflammation in the CNS and inhibits T cell-mediated lysis of glial target cells.
- Intranasal Delivery of Neural Stem Cells: A CNS-specific, Non-invasive Cell-based Therapy for Experimental Autoimmune Encephalomyelitis.
- Intravenous transfer of apoptotic cell-treated dendritic cells leads to immune tolerance by blocking Th17 cell activity.
- Immune tolerance induced by intravenous transfer of immature dendritic cells via up-regulating numbers of suppressive IL-10(+) IFN-? (+)-producing CD4 (+) T cells.
- Low dose zymosan ameliorates both chronic and relapsing experimental autoimmune encephalomyelitis.
- Generation of large numbers of highly purified dendritic cells from bone marrow progenitor cells after co-culture with syngeneic murine splenocytes.
- TCR stimulation upregulates MS4a4B expression through induction of AP-1 transcription factor during T cell activation.
- Intravenous tolerance effectively overcomes enhanced pro-inflammatory responses and experimental autoimmune encephalomyelitis severity in the absence of IL-12 receptor...
- CNS-specific Therapy for Ongoing EAE by Silencing IL-17 Pathway in Astrocytes.
- Magnetic resonance imaging of the cauda equina in Guillain-Barré syndrome.
- Guillain-Barré syndrome: clinical and immunological aspects.
- Search for antibodies to galactocerebroside in the serum and cerebrospinal fluid in human demyelinating Disorders.
- Plasmapheresis in acute attacks of Multiple sclerosis: the cons.
- Urinary free kappa light chain levels in chronic progressive Multiple sclerosis.
- Anti-interleukin-12 antibody: potential role in preventing relapses of Multiple sclerosis.
- Free light chains in Multiple sclerosis urine.
- Autoantigen-reactive T cells in neuroimmunology.
- Serial evoked potential studies and MRI imaging in chronic progressive Multiple sclerosis.
- P2-reactive T cells in inflammatory demyelination of the Peripheral nerve.
- Chemokines and Peripheral nerve demyelination.
- Chemokines and chemokine receptors in the pathogenesis of Multiple sclerosis.
- Role of the innate immune System in autoimmune inflammatory demyelination.
- Upregulation of chemokine receptor expression by IL-10/IL-4 in adult neural stem cells.
- Pathogenesis of neuroimmunologic Diseases. Experimental models.
- Pathophysiology of interleukin-23 in experimental autoimmune encephalomyelitis.
- A double-blind, placebo-controlled trial of extracorporeal photopheresis in chronic progressive Multiple sclerosis.
- IL-12 reverses the suppressive effect of the CD40 ligand blockade on experimental autoimmune encephalomyelitis (EAE).
- The protease inhibitor, Bowman-Birk Inhibitor, suppresses experimental autoimmune encephalomyelitis: a potential oral therapy for Multiple sclerosis.
- Retinal ganglion cell damage induced by spontaneous autoimmune Optic neuritis in MOG-specific TCR transgenic mice.
- IL-12R beta 2 promotes the development of CD4+CD25+ regulatory T cells.
- CD11c+CD11b+ dendritic cells play an important role in intravenous tolerance and the suppression of experimental autoimmune encephalomyelitis.
- Inflammatory demyelination induces axonal injury and retinal ganglion cell apoptosis in experimental Optic neuritis.
- Bowman-Birk inhibitor suppresses autoimmune inflammation and neuronal loss in a mouse model of Multiple sclerosis.
- Inducible IL-23p19 expression in human microglia via p38 MAPK and NF-kappaB signal pathways.
- Antigen presenting cells treated in vitro by macrophage colony-stimulating factor and autoantigen protect mice from autoimmunity.
- Suppression of autoimmune inflammation of the central Nervous system by interleukin 10 secreted by interleukin 27-stimulated T cells.
- Suppressive effect of IL-27 on encephalitogenic Th17 cells and the effector phase of experimental autoimmune encephalomyelitis.
- SIRT1 activation confers neuroprotection in experimental Optic neuritis.
- Effect of DAB(389)IL-2 immunotoxin on the course of experimental autoimmune encephalomyelitis in Lewis rats.
- The PD-1/PD-L pathway is up-regulated during IL-12-induced suppression of EAE mediated by IFN-gamma.
- Generation of immunogenic and tolerogenic clinical-grade dendritic cells.
- Bowman-Birk Inhibitor attenuates experimental autoimmune encephalomyelitis by delaying infiltration of inflammatory cells into the CNS.
- IL-9 is important for T-cell activation and differentiation in autoimmune inflammation of the central Nervous system.
- Kit (W-sh) mice develop earlier and more severe experimental autoimmune encephalomyelitis due to absence of immune suppression.
- The encephalitogenicity of T(H)17 cells is dependent on IL-1- and IL-23-induced production of the cytokine GM-CSF.
- Oral resveratrol reduces neuronal damage in a model of Multiple sclerosis.
- 3G11 expression in CD4+ T cell-mediated autoimmunity and immune tolerance.
- MS4a4B, a CD20 homologue in T cells, inhibits T cell propagation by modulation of cell cycle.
- IDO upregulates regulatory T cells via tryptophan catabolite and suppresses encephalitogenic T cell responses in experimental autoimmune encephalomyelitis.
- Neutralization of IL-9 ameliorates experimental autoimmune encephalomyelitis by decreasing the effector T cell population.
- Evaluation of bone marrow- and Brain-derived neural stem cells in therapy of central nervous system autoimmunity.
- Targeting ganglioside epitope 3G11 on the surface of CD4+ T cells suppresses EAE by altering the Treg/Th17 cell balance.
- Expression of 3G11 epitope defines subpopulations of regulatory T cells with different suppressive potency.
- T-cell based immunotherapy in experimental autoimmune encephalomyelitis and Multiple sclerosis.
- The TLR7 agonist, imiquimod, increases IFN-beta production and reduces the severity of experimental autoimmune encephalomyelitis.
- Current views on the roles of Th1 and Th17 cells in experimental autoimmune encephalomyelitis.
- Suppression of murine experimental autoimmune encephalomyelitis by interleukin-2 receptor targeted fusion toxin, DAB(389)IL-2.
- IL-9: basic biology, signaling pathways in CD4+ T cells and implications for autoimmunity.
- Timing of corticosteroid therapy is critical to prevent retinal ganglion cell loss in experimental Optic neuritis.
- Adult neural stem cells expressing IL-10 confer potent immunomodulation and remyelination in experimental autoimmune encephalitis.
- Differential effect of IL-27 on developing versus committed Th17 cells.
- IL-23 drives pathogenic IL-17-producing CD8+ T cells.
- Therapeutic potential of IL-27 in Multiple sclerosis?
- MOG(35-55) i.v suppresses experimental autoimmune encephalomyelitis partially through modulation of Th17 and JAK/STAT pathways.
- Intravenous tolerance modulates macrophage classical activation and antigen presentation in experimental autoimmune encephalomyelitis.
- IDO: a double-edged sword for T(H)1/T(H)2 regulation.
- Cellular remyelinating therapy in Multiple sclerosis.
- Magnetic cell sorting: a fast and effective method of concurrent isolation of high purity viable astrocytes and microglia from neonatal mouse Brain tissue.
- Cutting Edge: TLR3 stimulation suppresses experimental autoimmune encephalomyelitis by inducing endogenous IFN-beta.
- Retinal ganglion cell loss induced by acute optic neuritis in a relapsing model of Multiple sclerosis.
- Increased IL-23p19 expression in Multiple sclerosis lesions and its induction in microglia.
- Dendritic cells transduced with SOCS-3 exhibit a tolerogenic/DC2 phenotype that directs type 2 Th cell differentiation in vitro and in vivo.
- Loss of the surface antigen 3G11 characterizes a distinct population of anergic/regulatory T cells in experimental autoimmune encephalomyelitis.
- Glucosamine abrogates the acute phase of experimental autoimmune encephalomyelitis by induction of Th2 response.
- Astrocytes as antigen-presenting cells: expression of IL-12/IL-23.
- IL-27 subunits and its receptor (WSX-1) mRNAs are markedly up-regulated in inflammatory cells in the CNS during experimental autoimmune encephalomyelitis.
- A paradoxical role of APCs in the induction of intravenous tolerance in experimental autoimmune encephalomyelitis.
- Role of the IL-12/IL-23 system in the regulation of T-cell responses in central Nervous system inflammatory demyelination.
- Early administration of IL-12 suppresses EAE through induction of interferon-gamma.
- T cell and antibody responses in remitting-relapsing experimental autoimmune encephalomyelitis in (C57BL/6 x SJL) F1 mice.
- Role of IL-12 receptor beta 1 in regulation of T cell response by APC in experimental autoimmune encephalomyelitis.
- Differential expression and regulation of IL-23 and IL-12 subunits and receptors in adult mouse microglia.
- Skeletal muscle myosin is the autoantigen for experimental autoimmune myositis.
- Induction of experimental autoimmune encephalomyelitis in IL-12 receptor-beta 2-deficient mice: IL-12 responsiveness is not required in the pathogenesis of inflammator...
Schools
Pahlavi University
Procedures Preformed
- EEG (Electroencephalogram)
Conditions Treated
- Alzheimer's Disease
- Brain and Nervous System Cancer (incl. Gliomas, Astrocytoma, Schwannoma, Medulloblastoma, Chordoma)
- Convulsions
- Dementia
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Doctors Specialties
- Neurology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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University of Pennsylvania Health System -
Shiraz University of Medical Sciences
Hospital
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Thomas Jefferson University Hospital
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