Associate Professor of Pediatrics, Massachusetts General Hospital

Pediatrics

Bobby Cherayil is an expert in the field of Pediatrics. He graduated from Christian Medical College Vellore. Bobby Cherayil is a published doctor as well. He has 33 publications published. The latest was: Commensal bacteria-induced interleukin-1β (IL-1β) secreted by macrophages upregulates hepcidin expression in hepatocytes by activating the bone morphogenetic protein s...

Publications

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• Commensal bacteria-induced interleukin-1β (IL-1β) secreted by macrophages upregulates hepcidin expression in hepatocytes by activating the bone morphogenetic protein s...
• Colitis Induces Enteric Neurogenesis Through a 5-HT4-dependent Mechanism.
• Effect of Human Immunodeficiency Virus Infection on Plasma Bactericidal Activity against Salmonella enterica Serovar Typhimurium.
• Intestinal Inflammation Modulates Expression of the Iron-Regulating Hormone Hepcidin Depending on Erythropoietic Activity and the Commensal Microbiota.
• Co-infection with an intestinal helminth impairs host innate immunity against Salmonella and exacerbates intestinal inflammation in mice.
• Serum-induced up-regulation of hepcidin expression involves the bone morphogenetic protein signaling pathway.
• Role of anti-lipopolysaccharide antibodies in serum bactericidal activity against Salmonella enterica serovar Typhimurium in healthy adults and children in the United ...
• Increased susceptibility of melanin-concentrating hormone-deficient mice to infection with Salmonella enterica serovar Typhimurium.
• Helminth Infection Impairs Autophagy-Mediated Killing of Bacterial Enteropathogens by Macrophages.
• Tumor necrosis factor α inhibits expression of the iron regulating hormone hepcidin in murine models of innate colitis.
• Iron and intestinal immunity.
• The role of iron in the immune response to bacterial infection.
• Regulation of Lipopolysaccharide-Induced Translation of Tumor Necrosis Factor-Alpha by the Toll-Like Receptor 4 Adaptor Protein TRAM.
• The bone morphogenetic protein-hepcidin axis as a therapeutic target in inflammatory bowel disease.
• Role of ferroportin in macrophage-mediated immunity.
• Identification of Drosophila Yin and PEPT2 as evolutionarily conserved phagosome-associated muramyl dipeptide transporters.
• Cross-talk between iron homeostasis and intestinal inflammation.
• Selective modulation of TLR4-activated inflammatory responses by altered iron homeostasis in mice.
• Indoleamine 2,3-dioxygenase in intestinal immunity and inflammation.
• Attenuated inflammatory responses in hemochromatosis reveal a role for iron in the regulation of macrophage cytokine translation.
• Deficiency of indoleamine 2,3-dioxygenase enhances commensal-induced antibody responses and protects against Citrobacter rodentium-induced colitis.
• Alternatively activated macrophages in intestinal helminth infection: effects on concurrent bacterial colitis.
• Intestinal innate immunity and the pathogenesis of Salmonella enteritis.
• The iron efflux protein ferroportin regulates the intracellular growth of Salmonella enterica.
• Perisinusoidal B cells in the bone marrow participate in T-independent responses to blood-borne microbes.
• Developmentally regulated intestinal expression of IFN-gamma and its target genes and the age-specific response to enteric Salmonella infection.
• Toll-like receptor 4 mutation impairs the macrophage TNFalpha response to peptidoglycan.
• Cooperative interactions between flagellin and SopE2 in the epithelial interleukin-8 response to Salmonella enterica serovar typhimurium infection.
• Developmentally regulated IkappaB expression in intestinal epithelium and susceptibility to flagellin-induced inflammation.
• Dispatch from India.
• How not to get bugged by bugs: mechanisms of cellular tolerance to microorganisms.
• Role of Toll-like receptor 4 in macrophage activation and tolerance during Salmonella enterica serovar Typhimurium infection.

Doctors Specialties

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• Pediatrics

Accepted Insurances

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Education

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• New York Presbyterian Hospital - Columbia Campus
• Christian Medical College Vellore

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